Dormancy induction - Revision history

Datuerff at 18:32, 18 April 2013

2013-04-18T18:32:42Z

Datuerff at 18:28, 18 April 2013

2013-04-18T18:28:02Z

Datuerff at 18:24, 18 April 2013

2013-04-18T18:24:14Z

Datuerff at 18:20, 18 April 2013

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Datuerff at 18:18, 18 April 2013

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Datuerff at 18:15, 18 April 2013

2013-04-18T18:15:36Z

Datuerff at 19:58, 28 February 2013

2013-02-28T19:58:08Z

Datuerff at 19:35, 28 February 2013

2013-02-28T19:35:37Z

Datuerff at 19:05, 28 February 2013

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Datuerff at 17:03, 26 February 2013

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 PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor
-DAM genes, Evergrowing peach: [http://www.ncbi.nlm.nih.gov/pubmed/21899556] [http://jhered.oxfordjournals.org/content/95/5/436.long]
+DAM genes, Evergrowing ''Prunus persica'': [http://www.ncbi.nlm.nih.gov/pubmed/21899556] [http://jhered.oxfordjournals.org/content/95/5/436.long][http://www.ncbi.nlm.nih.gov/pubmed/21378115]
 SSRs: [[File:All_primers.docx]]

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 PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor
-DAM genes:
+DAM genes, Evergrowing peach: [http://www.ncbi.nlm.nih.gov/pubmed/21899556] [http://jhered.oxfordjournals.org/content/95/5/436.long]
 SSRs: [[File:All_primers.docx]]

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 "ABI3, FUS3 and LEC2 encode related plant-specific transcription factors containing the conserved B3 DNA binding domain (Giraudat et al., 1992; Luerssen et al., 1998; Stone et al., 2001), whereas LEC1 encodes a HAP3 subunit of the CCAAT binding transcription factor (CBF, also known as NF-Y (Lotan et al., 1998)). All four abi3, lec1, lec2 and fus3 mutants are severely affected in seed maturation and share some common phenotypes, such as decreased dormancy at maturation (Raz et al., 2001) and reduced expression of seed storage proteins" - [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3243337/]
 SUA - splicing factor, splices ABI3- [http://www.ncbi.nlm.nih.gov/pubmed/?term=SUA%20splicing%20factor]
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 "It is clear from the results that glucose delays germination by activating the ABA signalling pathway via ABI3 and repressing the GA signalling pathway via SPY and RGL2. It has been shown that glucose application has a pronounced effect on ABI3 and RGL2, up-regulating both at the transcriptional level."
 GAI Della protein [Arabidopsis thaliana] Member of the DELLA proteins that restrain the cell proliferation and expansion that drives plant growth. KEGG - [http://www.genome.jp/dbget-bin/www_bget?ath:AT1G14920]
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 ERF4- ethylene-responsive transcription factor 4 - [http://www.genome.jp/dbget-bin/www_bget?ath:AT3G15210]
 PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor
-SSRs
+DAM genes:
-[[File:All_primers.docx]]
+SSRs: [[File:All_primers.docx]]

← Older revision		Revision as of 18:20, 18 April 2013
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	SSRs		SSRs
		+
		+	[[File:All_primers.docx]]

← Older revision		Revision as of 18:18, 18 April 2013
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	"ABI3, FUS3 and LEC2 encode related plant-specific transcription factors containing the conserved B3 DNA binding domain (Giraudat et al., 1992; Luerssen et al., 1998; Stone et al., 2001), whereas LEC1 encodes a HAP3 subunit of the CCAAT binding transcription factor (CBF, also known as NF-Y (Lotan et al., 1998)). All four abi3, lec1, lec2 and fus3 mutants are severely affected in seed maturation and share some common phenotypes, such as decreased dormancy at maturation (Raz et al., 2001) and reduced expression of seed storage proteins" - [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3243337/]		"ABI3, FUS3 and LEC2 encode related plant-specific transcription factors containing the conserved B3 DNA binding domain (Giraudat et al., 1992; Luerssen et al., 1998; Stone et al., 2001), whereas LEC1 encodes a HAP3 subunit of the CCAAT binding transcription factor (CBF, also known as NF-Y (Lotan et al., 1998)). All four abi3, lec1, lec2 and fus3 mutants are severely affected in seed maturation and share some common phenotypes, such as decreased dormancy at maturation (Raz et al., 2001) and reduced expression of seed storage proteins" - [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3243337/]

−	~~[[File:f6.large.jpg]]~~

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-Dormancy is an adaptation mechanism that is vital to the survival of plants.  Plant dormancy is induced by hormones such as abscisic acid.  While it has many uses, abscisic acid inhibits fruit ripening, inhibits cell growth/seed germination, and downregulates enzymes needed for photosynthesis.
+Dormancy is an essential survival mechanism for most plants.  During this phase, plants enter into a state of suspended growth.  Environmental factors such as lack of water or extreme temperature change can induce this state until conditions are favorable again. Dormancy pertains to agriculture with the importance of keeping crops alive during harsh weather.
 [[File:dormancygenes.png]]
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 ERF4- ethylene-responsive transcription factor 4 - [http://www.genome.jp/dbget-bin/www_bget?ath:AT3G15210]
-DOG1 is expressed in seeds of these two species although they have only a shallow dormancy. LesaDOG1 transcript expression in L. sativum seed tissues (radicle and micropylar endosperm) is regulated by imbibition and is '''ABA-inducible'''.
+PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor
-PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor
+SSRs

← Older revision		Revision as of 19:58, 28 February 2013
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	PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor		PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor
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−	In seed dormancy, how do DOG1 and the ABA pathway interact with eachother? Is there one pathway that is always active during dormancy, or there entirely separate genes for dormancy in seeds versus dormancy after germination?

← Older revision		Revision as of 19:35, 28 February 2013
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−	In seed dormancy, how do DOG1 and the ABA pathway interact with eachother? ~~Do they interact~~, or ~~are~~ there entirely separate genes for dormancy in seeds versus dormancy after germination?	+	In seed dormancy, how do DOG1 and the ABA pathway interact with eachother? Is there one pathway that is always active during dormancy, or there entirely separate genes for dormancy in seeds versus dormancy after germination?

← Older revision		Revision as of 19:05, 28 February 2013
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	PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor		PYR/PYL/RCAR - [http://www.ncbi.nlm.nih.gov/pubmed/23265948] - ABA receptor
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		+
		+	In seed dormancy, how do DOG1 and the ABA pathway interact with eachother? Do they interact, or are there entirely separate genes for dormancy in seeds versus dormancy after germination?

← Older revision		Revision as of 17:03, 26 February 2013
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	Plant Hormone Signal Induction in Arabidopsis thaliana: [http://www.genome.jp/kegg-bin/show_pathway?ath04075][[File:ath0475.png]]		Plant Hormone Signal Induction in Arabidopsis thaliana: [http://www.genome.jp/kegg-bin/show_pathway?ath04075][[File:ath0475.png]]
		+
		+	Ethylene- breaks dormancy. ''etr1 and ein 2'' mutants displayed increased dormancy which relates to increased sensitivity to ABA. ''ctr1'' mutations led to decreased ABA sensitivity. [http://www.annualreviews.org/doi/full/10.1146/annurev.arplant.59.032607.092740?url_ver=Z39.88-2003&rfr_id=ori:rid:crossref.org&rfr_dat=cr_pub%3dpubmed]

	Gene expression during the induction, maintenance, and release of dormancy in apical buds of poplar . From this paper: "Three regulatory genes, AP2/EREBP, ERF4, and WRKY11, and one expressed protein gene were associated with this crucial step in dormancy induction (Fig. 7). Based on the function of the respective Arabidopsis homologues, the three regulatory genes could act downstream of ethylene and/or abscisic acid (ABA) signals during dormancy induction." [http://jxb.oxfordjournals.org/content/58/15-16/4047.full]		Gene expression during the induction, maintenance, and release of dormancy in apical buds of poplar . From this paper: "Three regulatory genes, AP2/EREBP, ERF4, and WRKY11, and one expressed protein gene were associated with this crucial step in dormancy induction (Fig. 7). Based on the function of the respective Arabidopsis homologues, the three regulatory genes could act downstream of ethylene and/or abscisic acid (ABA) signals during dormancy induction." [http://jxb.oxfordjournals.org/content/58/15-16/4047.full]